Spinal cord stimulation and differential target multiplexed programming

Presenter and Advisor Information

João Pedro Mezzena, Illinois Wesleyan University

Major

Psychology

Submission Type

Poster

Area of Study or Work

Neuroscience, Psychology

Faculty Advisor

Joe Williams

Expected Graduation Date

2027

Location

CNS Atrium

Start Date

4-12-2025 8:30 AM

End Date

4-12-2025 9:30 AM

Abstract

Spinal cord stimulation (SCS) is a treatment for people who suffer chronic pain conditions, such as chronic back pain. SCS involves implanting an electrode directly into the spinal cord to send electrical impulses. This treatment has proven to be clinically effective for people who have been suffering with pain. However, the continuous use of (SCS) has shown some issues such as faster battery depletion which could ultimately decrease the efficacy of the SCS treatment. In this research, we are testing an alternate method using two differential target multiplexed programming (DTMP) models to determine whether the new methodology is 1) as efficacious and 2) works via a different biological mechanism than the continuous low-rate SCS treatment. DTMP treatments significantly decreased mechanical hypersensitivity in the SNI animal model of neuropathic pain, with the DTMP models having a significantly higher efficacy in pain reduction in comparison to continuous low-rate SCS. In addition, the DTMP models modulate different genes than the continuous low-rate SCS treatment, indicating that DTMP works by a different biological mechanism.

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Apr 12th, 8:30 AM Apr 12th, 9:30 AM

Spinal cord stimulation and differential target multiplexed programming

CNS Atrium

Spinal cord stimulation (SCS) is a treatment for people who suffer chronic pain conditions, such as chronic back pain. SCS involves implanting an electrode directly into the spinal cord to send electrical impulses. This treatment has proven to be clinically effective for people who have been suffering with pain. However, the continuous use of (SCS) has shown some issues such as faster battery depletion which could ultimately decrease the efficacy of the SCS treatment. In this research, we are testing an alternate method using two differential target multiplexed programming (DTMP) models to determine whether the new methodology is 1) as efficacious and 2) works via a different biological mechanism than the continuous low-rate SCS treatment. DTMP treatments significantly decreased mechanical hypersensitivity in the SNI animal model of neuropathic pain, with the DTMP models having a significantly higher efficacy in pain reduction in comparison to continuous low-rate SCS. In addition, the DTMP models modulate different genes than the continuous low-rate SCS treatment, indicating that DTMP works by a different biological mechanism.